Caro Pierre, complimenti per aver postato due volte lo stesso studio sulle donne svedesi da due link diversi, il che vuol dire che nemmeno leggi quello che posti.
Se avessi letto avresti anche appreso che questo studio si base sulla compilazione di un questionario relativo alle abitudini alimentari.
Ti ricorda qualcosa? a me ricorda tanto il medico USA che pretendeva di dimostrare l'esistenza della sindrome post finasteride facendo compilare un questionario ai partecipanti.
Tu le vedi le similitudini, Ceongrad?
Ma tornando a te Pierre.
Lo studio postato sui presunti danni renali conclude:
To conclude, it appears that protein intake under 2. 8 g.kg does not impair renal function in well-trained athletes as indicated by the measures of renal function used in this study
C'e' bisogno di tradurti o capisci da te che vuol dire?
Inoltre, nel corpo dell'abastract c'e' ulteriormente evidenziato:
The data revealed that despite higher plasma concentration of uric acid and calcium, Group BB had renal clearances of creatinine, urea, and albumin that were within the normal range. The nitrogen balance for both groups became positive when daily protein intake exceeded 1.26 g.kg but there were no correlations between protein intake and creatinine clearance, albumin excretion rate, and calcium excretion rate
Vuoi una traduzione ed una spiegazione di cosa significhi da un punto di vista medico o ci arrivi da solo?
Sulla bellezza dei carboidrati:
http://ajcn.nutrition.org/content/80/5/1175.full
a higher saturated fat intake was associated with a smaller decline in mean minimal coronary diameter (P = 0.001) and less progression of coronary stenosis (P = 0.002) during follow-up.
Carbohydrate intake was positively associated with atherosclerotic progression (P = 0.001), particularly when the glycemic index was high.
Polyunsaturated fat intake was positively associated with progression when replacing other fats (P = 0.04) but not when replacing carbohydrate or protein.
Sulla presunta pericolosita' di una dietalow carb:
A low carbohydrate Mediterranean diet improves cardiovascular risk factors and diabetes control among overweight patients with type 2 diabetes mellitus: a 1-year prospective randomized intervention study.
Elhayany A, Lustman A, Abel R, Attal-Singer J, Vinker S.
Meir Medical Center, Kfar Saba, Israel. elasher@clalit.org.il
Abstract
BACKGROUND: The appropriate dietary intervention for overweight persons with type 2 diabetes mellitus (DM2) is unclear. Trials comparing the effectiveness of diets are frequently limited by short follow-up times and high dropout rates. AIM: The effects of a low carbohydrate Mediterranean (LCM), a traditional Mediterranean (TM), and the 2003 American Diabetic Association (ADA) diet were compared, on health parameters during a 12-month period. METHODS: In this 12-month trial, 259 overweight diabetic patients (mean age 55 years, mean body mass index 31.4 kg/m(2)) were randomly assigned to one of the three diets. The primary end-points were reduction of fasting plasma glucose, HbA1c and triglyceride (TG) levels. RESULTS: 194 patients out of 259 (74.9%) completed follow-up. After 12 months, the mean weight loss for all patients was 8.3 kg: 7.7 kg for ADA, 7.4 kg for TM and 10.1 kg for LCM diets. The reduction in HbA1c was significantly greater in the LCM diet than in the ADA diet (-2.0 and -1.6%, respectively, p < 0.022). HDL cholesterol increased (0.1 mmol/l +/- 0.02) only on the LCM (p < 0.002). The reduction in serum TG was greater in the LCM (-1.3 mmol/l) and TM (-1.5 mmol/l) than in the ADA (-0.7 mmol/l), p = 0.001. CONCLUSIONS: An intensive 12-month dietary intervention in a community-based setting was effective in improving most modifiable cardiovascular risk factors in all the dietary groups. Only the LCM improved HDL levels and was superior to both the ADA and TM in improving glycaemic control.
Necessita una spiegazione o si puo' comprendere da soli?
Inoltre: lo studio parla di low carbohydrate Mediterranean diet, come e' possibile? La dieta mediterannea a basso tenore di carboidrati???
Altro studio interessante:
http://www.meatsafety.org/ht/a/GetDocumentAction/i/47784
Qui le percentuali di macronutrienti sono 40-30-30.
La proporzione e' uguale alla zona e viene definita come A Moderate-Protein Diet, moderate...
L'altro gruppo seguiva una dieta con le seguenti proporizioni di macronutrienti: 55% carboidrati, 15% proteine, 30% grassi.
Vi ricorda qualcosa? sembra la dieta che propone il buon Calabrese...
Comunque, queste sono le conclusioni:
This study demonstrates the merit of a moderate PRO diet
for weight loss and long-term weight maintenance. The DRI
guidelines allow for diets with diverse macronutrient ratios but provide no guidance on how to apply the AMDR to specific
health conditions or individual lifestyles. The findings of the
current study demonstrate that although energy deficit is the
major factor for body weight loss, the macronutrient composition
affects body composition, blood lipids, and long-term
compliance. Specifically, a PRO diet with protein at the upper
end and carbohydrates at the lower end of the AMDR is more
effective for reducing %Fat and improving dyslipidemia...
Sulla presunta correlazione tra proteine, acidita' e decalcificazione:
http://cdn.intechweb.org/pdfs/26682.pdf
Queste le conclusioni:
Protein acts on bone metabolism at different levels and through different mechanisms.
There is little evidence that a high-protein diet will increase bone loss. Protein is well-known to be calciuric, yet there are conflicting data on whether the excreted calcium comes from an increase of calcium absorption or from bone resorption. The direct effects of protein on bone turnover markers and BMD seem to be positive when considering observational studies, but interventional studies do not provide significant outcomes to conclude. Finally, when considering fracture rate, there seems to be a small positive effect of protein on bone as long as calcium levels remain adequate.
Two mechanisms are proposed to explain the action of protein on bone: the acid-ash theory and the hormonal anabolic effect through IGF-1 and CaSR. The hormonal anabolic mechanism supports the fact that protein is beneficial to bone by increasing IGF-1. On the other hand, the acid-ash theory considers that the acid load due to protein consumption is harmful to bone. If both mechanisms occur at the same time, it is possible to benefit from the protein-induced IGF-1 without the negative effect of the acid load by compensating the diet with adequate alkalinizing foods.
Dietary protein quality adds complexity to the protein debate. It has been hypothesized that animal protein would be more deleterious to bone than vegetal protein. However, studies show no real difference between those two protein sources
Ciao Pierre, hai perso un'altra occasione per evitare di fare una magra figura.
Purtroppo, troppo spesso, ignoranza fa rima con arroganza.
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