Cosa ne pensate di questo estratto preso dall'autorevole sito hairlosstalk;
Trattasi di studio scientifico che da' ulteriore valore al concetto di vascolarizzazione aumentata in funzione dell'aumento dei vasi nello scalpo.....che porta all'ingrossamento del follicolo.
This study examined tissues from the transgenic high VEGF expressing mice for the extent of angiogenesis around hair follicles, measured the size of the hair follicles, and compared these statistics with those from normal mice. In addition, the researchers injected an antibody that blocks VEGF activity into normal mice and also exposed cultured hair follicle cells to VEGF. They found that the transgenic mice had significantly larger anagen hair follicles in association with VEGF production and angiogenesis as compared to normal mice. Blocking VEGF activity resulted in a delay of hair follicles switching from telogen to anagen and much smaller anagen hair follicles developed. Their culture studies showed that VEGF had no direct stimulatory effect on hair follicle growth. That is, it was the formation of blood vessels that allowed increased hair follicle activity in the mutated mice and not any direct effect on the hair follicle cells by the VEGF signal. The study concludes that VEGF has an important role in controlling hair biology and that hair follicle size is partly dependant on VEGF induced angiogenesis.
daje che se po' fa'
Trattasi di studio scientifico che da' ulteriore valore al concetto di vascolarizzazione aumentata in funzione dell'aumento dei vasi nello scalpo.....che porta all'ingrossamento del follicolo.
This study examined tissues from the transgenic high VEGF expressing mice for the extent of angiogenesis around hair follicles, measured the size of the hair follicles, and compared these statistics with those from normal mice. In addition, the researchers injected an antibody that blocks VEGF activity into normal mice and also exposed cultured hair follicle cells to VEGF. They found that the transgenic mice had significantly larger anagen hair follicles in association with VEGF production and angiogenesis as compared to normal mice. Blocking VEGF activity resulted in a delay of hair follicles switching from telogen to anagen and much smaller anagen hair follicles developed. Their culture studies showed that VEGF had no direct stimulatory effect on hair follicle growth. That is, it was the formation of blood vessels that allowed increased hair follicle activity in the mutated mice and not any direct effect on the hair follicle cells by the VEGF signal. The study concludes that VEGF has an important role in controlling hair biology and that hair follicle size is partly dependant on VEGF induced angiogenesis.
daje che se po' fa'